My Recovery Audit
Check every item that applies to your current daily life and injury history. The brain after TBI is operating at a lower threshold — the same inputs that trigger seizures in a sensitized brain also block recovery in an injured one.
Injury History
This is my first documented TBI or concussion
I have had 2 or more concussions (cumulative injury changes recovery trajectory)
My most recent injury was less than 3 months ago (acute phase)
I am still symptomatic more than 3 months after my injury (post-concussion syndrome)
I have a history of whiplash, neck injury, or forceful impact to the upper spine
My injury involved loss of consciousness, even briefly
I was not given specific recovery instructions beyond "rest for a few days"
I returned to screen use, work, or school within 48–72 hours of injury
I have had a difficult birth (forceps, vacuum, prolonged labor) — relevant to cranial structure and baseline resilience
Current Symptoms — Check All That Apply
These are the documented effects of unresolved excitotoxicity, mitochondrial dysfunction, and neuroinflammation — the mechanisms that sustain post-concussion syndrome. They are also the symptoms most directly affected by the inputs in this audit.
Persistent headache or head pressure
Light sensitivity (photophobia)
Sound sensitivity (phonophobia)
Cognitive fatigue / brain fog
Difficulty concentrating or word-finding
Memory problems (short-term)
Sleep disturbance — difficulty falling or staying asleep
Mood dysregulation — irritability, anxiety, depression
Balance or vestibular issues
Visual disturbance — blurring, tracking difficulty, eye fatigue
Neck pain or occipital tightness
Barometric pressure sensitivity — symptoms worsen before rain or storms
Symptom crash after screen use or cognitive effort ("PEM-like" pattern)
Fatigue disproportionate to activity level
The GABA Connection — Why TBI and Seizure Threshold Are the Same Problem
In seizure disorders, the core mechanism is insufficient GABA (inhibitory) relative to glutamate (excitatory). In TBI, the acute crisis is glutamate excess — the secondary injury cascade. But the sustained post-concussion problem is identical: GABAergic interneurons are preferentially destroyed by excitotoxic damage, leaving the brain with reduced inhibitory infrastructure. The same inputs that deplete GABA in a seizure disorder are blocking recovery here.
Post-concussion symptoms are largely GABAergic in origin. Anxiety, light sensitivity, sound sensitivity, sleep disruption, mood dysregulation, and cognitive fatigue — all documented in MRS (magnetic resonance spectroscopy) studies showing reduced GABA in visual cortex, prefrontal cortex, and hippocampus after concussion. These are not vague complaints. They are the measurable signatures of a brain operating without adequate inhibitory tone.
Post-traumatic epilepsy risk. TBI is one of the most significant risk factors for developing seizures. The excitotoxic damage from the injury — and from every repeat injury — lowers seizure threshold. Hemosiderin deposits (iron released from lysed red blood cells after any brain bleed, including microbleeds from concussion) generate free radicals indefinitely through Fenton chemistry, creating ongoing cortical irritation. Every head injury that caused bleeding deposits iron into cortical tissue. This is the documented mechanism behind delayed-onset post-traumatic epilepsy. The inputs in this audit are relevant on two levels: recovery from TBI and protection against post-traumatic seizures.
What depletes GABA — same in both TBI and seizures
· Fluoride → inhibits GAD (glutamate→GABA enzyme)
· B6 depletion (OCs, antibiotics, processed food) → GAD cofactor
· Gut dysbiosis → destroys GABA-producing bacteria
· PFAS thyroid disruption → reduces GABA receptor density
· Benzodiazepine tolerance → GABA receptor downregulation
· Aspartame → excitatory load; blocks inhibitory rebound
What GABA deficiency looks like post-TBI
· Anxiety and nervous system hyperreactivity
· Light and sound sensitivity (lowered sensory threshold)
· Sleep onset difficulty and fragmented sleep
· Mood dysregulation / irritability
· Cognitive fatigue disproportionate to effort
· Symptom crash after sensory or cognitive load
Water
I drink fluoridated municipal tap water
I drink filtered water but don't know if the filter removes fluoride
I drink distilled or reverse osmosis water (mineral-stripped — leaches minerals from the body)
I drink from aluminum cans regularly (soda, sparkling water, energy drinks)
I rarely drink plain water throughout the day — dehydration is a significant TBI recovery barrier
Goal: natural spring water (findaspring.com) or non-ozonated bottled spring water · glass or stainless steel containers
Food, Diet & the Excitotoxin Load
After TBI, the brain releases massive amounts of glutamate from injured neurons — the secondary injury cascade. Adding dietary free glutamate to an already-flooded system extends and amplifies this damage. This is the part that doesn't make the discharge paperwork.
I use protein powders (whey, plant-based, collagen with additives) — high free glutamate; feeds secondary injury in recovering brain
I eat processed food daily — packaged, fast food, restaurant more than 3x/week
I use artificial sweeteners — aspartame (diet drinks, Equal), sucralose (Splenda), acesulfame-K
I eat foods with MSG, hydrolyzed protein, yeast extract, or "natural flavors" regularly
I eat conventional (non-organic) produce from the Dirty Dozen list — pyrethroids affect sodium channels
I eat conventional meat, dairy, or farmed fish (subtherapeutic antibiotics destroy GABA-producing gut bacteria)
I skip meals or go more than 4–5 hours without eating — glucose instability impairs neuronal repair
I eat artificial food dyes daily (brightly colored snacks, cereals, drinks)
My child (or I) eat commercially produced candy regularly — gummies, sour candy, hard candy, chocolate
Florida DOH and Lead Safe Mama testing identified lead, arsenic, and cadmium in popular commercial candy brands (Sour Patch Kids, Ring Pops, Nerds, Jolly Ranchers, Airheads, Trolli, Haribo, and others). Lead accumulates in the brain and has no safe threshold for neurological effect. For a brain already recovering from injury, heavy metal burden from daily candy consumption is a documented and unaddressed input.
Caffeine & Stimulants
I drink coffee, tea, or caffeinated beverages daily — adenosine (sleep drive) is critical for brain repair; caffeine blocks it
I use energy drinks (Monster, Red Bull, Celsius, Reign)
I use pre-workout supplements
I take OTC medications containing caffeine (Excedrin, Midol, Anacin, NoDoz)
Supplements & Personal Care
I take a daily multivitamin
Retinyl palmitate (synthetic vitamin A) raises intracranial pressure — directly contraindicated in the context of TBI. Synthetic D3 raises serum calcium, activating voltage-gated calcium channels. Folic acid (not food-form folate) blocks the folate receptor in MTHFR variants and masks B12 deficiency. TiO₂ coating on the tablet — titanium dioxide, flagged for neurological effects. None of these are what the label implies.
I supplement with Vitamin D (D3 capsules or drops) — soft tissue calcification risk; use morning sunlight and food sources (cod liver oil, fatty fish, pastured egg yolk)
I use fluoride toothpaste (most commercial brands)
Fluoride toothpaste is 1,000–1,500 ppm. Fluoride varnish applied at dental cleanings is 22,600 ppm — directly on oral mucosa. Fluoride inhibits glutamate decarboxylase (GAD), the enzyme that converts glutamate to GABA. For a recovering brain, every daily fluoride exposure is suppressing GABA synthesis at the enzymatic level.
I take OTC antihistamines, pain relievers, or cold medicine regularly
I have taken a fluoroquinolone antibiotic (Cipro, Levaquin, Avelox) in the past 2 years
Cookware & Food Packaging
I cook with nonstick (Teflon/PTFE) pans — especially on high heat
My nonstick pans have scratches or chips in the coating
I drink from aluminum cans regularly
I store or heat food in plastic containers
Bedroom & Home Environment
Non-native EMF activates voltage-gated calcium channels and generates reactive oxygen species — the same pathway that drives neuroinflammation in TBI. Sleep is the primary window for glymphatic clearance and synaptic repair. The bedroom environment determines the quality of that window.
My Wi-Fi router is on overnight (not on a timer)
I charge my phone on or near my nightstand
I have a smart TV, streaming stick, or other device in my bedroom
My smart meter is on a bedroom wall or within 15 feet of where I sleep
I can see a cell tower, power lines, or electrical infrastructure from my home
I have solar panels on or near the house (inverter creates dirty electricity)
I use LED lighting in my bedroom or main living areas in the evening
My bedroom is not fully dark at night
I use Bluetooth headphones or earbuds regularly (including while sleeping)
Screens & Cognitive Load
Screen use after TBI imposes metabolic demand on a brain that is already in energy deficit. Light sensitivity and post-exertion symptom crashes are signs that the recovery threshold is being exceeded.
I use screens within 1 hour of bedtime
I returned to full screen use (work, phone, TV) within 1 week of injury
My symptoms worsen after screen use — I use screens anyway
I game in a dark room or with high-contrast backlighting
I use wireless headphones during screen sessions
I have not been told the specific screen exposure thresholds relevant to TBI recovery
Sunlight & Sleep
Sleep is not passive rest after TBI — it is when glymphatic clearance of excess glutamate and metabolic waste occurs, when synaptic consolidation and repair happens, and when cortisol resets. Disrupting it extends every symptom.
I do not get outdoor morning sunlight within the first hour after waking
My first light exposure is a screen (phone, computer, TV)
I sleep fewer than 7–8 hours most nights
I wake with a dry mouth or have been told I snore or stop breathing
I sleep on my back — lateral sleep supports glymphatic flow
My sleep schedule is irregular — different bedtimes each night
I take sleep medication or antihistamines to sleep
Medications Taken
List your current medications. Check any flag items that apply.
Current medications
Supplements / OTC used regularly
Flag items — check any that apply:
I take acetaminophen (Tylenol) regularly for TBI headaches — depletes glutathione, the brain's primary antioxidant defense; the #1 cause of acute liver failure in the US
I take NSAIDs (ibuprofen, naproxen) regularly — GI and renal burden; magnesium interaction
I take a proton pump inhibitor (Prilosec, Nexium, Prevacid) — depletes magnesium, the brain's natural NMDA channel blocker
I take a benzodiazepine (Xanax, Valium, Ativan, Klonopin) — GABA receptor downregulation during tolerance; rebound excitability
I take oral contraceptives — deplete B6 and folate; B6 is required for GABA synthesis
I take corticosteroids (prednisone, methylprednisolone) — blood sugar dysregulation affects neuronal stability and repair
I take a daily antihistamine (Benadryl) — anticholinergic burden impairs cognitive recovery
I take iron supplements
Free iron generates hydroxyl radicals via Fenton chemistry — the same mechanism responsible for hemosiderin-related post-traumatic cortical irritation. Iron supplementation without confirmed deficiency adds to an oxidative environment in a brain already managing iron from microbleeds. Serum ferritin testing before supplementing is worth discussing with a provider.
I am currently taking or recently completed a course of antibiotics
I take medication for post-traumatic seizures (AED) — same nutrient depletion profile as seizure disorder; see Seizure Threshold Audit for full AED flag list
Antibiotics — History & Class
Not all antibiotics carry the same neurological risk. Check any class you have taken — recently or repeatedly in the past. Use this list to start a conversation with your prescriber.
Fluoroquinolone — Cipro, Levaquin, Avelox, Floxin, Factive, Noroxin, Baxdela
Published side effects: seizures (FDA Black Box Warning), confusion, hallucinations, dizziness, insomnia, tendon rupture, and peripheral neuropathy (which may be permanent). Also disrupts gut bacteria that produce GABA and neurotransmitter precursors. Discuss with your provider: Is there a lower-risk alternative given my TBI history?
Metronidazole / Tinidazole — Flagyl · Tindamax (same class, same risks)
Published side effects: seizures (listed in prescribing information), confusion, dizziness, headache, coordination problems, slurred speech, peripheral neuropathy, nausea, and metallic taste. Discuss with your provider: Is there an alternative for this infection that does not carry the same CNS risk?
Cephalosporin — 1st gen: Keflex (cephalexin), Duricef (cefadroxil), Ancef (IV) · 2nd gen: Ceftin, Cefzil, Ceclor · 3rd gen: Omnicef, Suprax, Vantin, Rocephin (injection) · 4th gen: Maxipime (IV)
Published side effects: seizures (listed in prescribing information), myoclonus (muscle jerking), encephalopathy, confusion, and dizziness. Risk increases if kidney function is impaired — cephalexin is primarily cleared by the kidneys and accumulates when clearance is reduced. Particularly relevant for a brain with lowered threshold post-TBI. Discuss with your provider: Has my kidney function been checked recently? Is there an alternative?
TMP-SMX — Bactrim, Septra
Published side effects: anemia, rash, nausea, and kidney effects. Can raise blood levels of certain medications — relevant if taking any neurological or psychiatric drugs. Discuss with your provider: Do I need medication level monitoring during this course?
Amoxicillin (Amoxil), doxycycline (Vibramycin, Doryx), azithromycin (Zithromax / Z-pack), clindamycin (Cleocin), nitrofurantoin (Macrobid, Macrodantin)
Lower neurological risk compared to the classes above. All antibiotics disrupt gut bacteria that produce GABA and neurotransmitter precursors. Discuss with your provider: What probiotic support is appropriate during and after this course?
I have taken 3 or more antibiotic courses in the past 12 months
I have never taken a probiotic after an antibiotic course
Dental Procedures & Anesthetics
A routine dental appointment involves several agents that directly affect brain threshold. For someone recovering from TBI, the mechanisms are the same as for seizure disorders — the fluoride and epinephrine burden is relevant to any neurologically compromised brain.
I received topical fluoride varnish (22,600 ppm — 15x toothpaste concentration) at my last dental cleaning
My local anesthetic contained epinephrine — published side effects include rapid heartbeat, elevated blood pressure, anxiety, headache; can elevate intracranial pressure and lower seizure threshold
I received nitrous oxide sedation — published side effects include nausea, headache, and with repeated use: B12 depletion leading to nerve damage and neurological decline
I have amalgam fillings that have not been safely removed
I have dental work planned and have not discussed my TBI history with my dentist
Before your next dental appointment
Print the TBI dental information sheet and bring it to your appointment. It covers epinephrine/ICP, fluoride varnish, nitrous/B12, cervical positioning, sensory sensitivity, amalgam, and the emergency protocol. Download: Dental Care & Anesthesia — TBI Patient Information Sheet
My audit summary — what stands out most:
My Action Plan
Organized by what to remove, what to add, and what to test. Start with the removes — reducing the daily neurological burden is the first step in any recovery.
Remove — Reduce Ongoing Burden
Excitotoxins — Primary Priority for TBI
Remove protein powders (whey, soy, pea, collagen with additives) — free glutamate feeds secondary injury
Remove aspartame, sucralose, acesulfame-K from all food and drink
Remove MSG, hydrolyzed protein, yeast extract, "natural flavors" in processed food
Remove energy drinks, pre-workout, and high-caffeine beverages
Remove artificial food dyes (Red 40, Yellow 5, Yellow 6, Blue 1)
Why this matters for TBI: The excitotoxic cascade following brain injury — the secondary injury process — is directly amplified by dietary free glutamate. This is the mechanism. Protein powders given to TBI patients in hospital and rehab settings are a documented example of feeding the very process that is extending injury.
Water & Cookware
Switch to natural spring water (findaspring.com) or non-ozonated bottled spring water
Switch to glass or stainless steel — no aluminum cans for daily beverages
Replace nonstick pans with cast iron, carbon steel, stainless steel, or glass
Personal Care
Switch toothpaste: no fluoride, no NHA, no SLS, no TiO₂ — tooth powder or verified clean brand
Remove synthetic multivitamins, Vitamin D3 supplements, folic acid
Check all OTC medications for hidden caffeine, artificial dyes, and sweeteners
Request epinephrine-free anesthetic and opt out of fluoride varnish at dental cleanings
Environment & Screens
Router off at night — use a smart plug timer
Phone charged in another room overnight — not on the nightstand
All screens removed from bedroom
Replace LED bedroom lighting with incandescent or low-flicker bulbs for evening use
Blackout curtains — no external light sources during sleep
Limit screen exposure in the acute recovery phase — stop before symptoms worsen
Add — Rebuild What Was Depleted
Daily Recovery Foundations
Morning sunlight: eyes open, outdoors, within 30–60 minutes of waking — regulates cortisol, melatonin, and circadian rhythm; all three govern repair windows
Lateral sleep position: left or right side — maximizes glymphatic clearance of excess glutamate and metabolic waste from the brain overnight
Consistent sleep/wake time: same time every day — circadian regularity governs the repair cycle
Bare feet on ground outdoors: grass, soil, sand — 10–20 minutes daily alongside morning sunlight
Gentle movement: walking (not intense exercise) — supports cerebral blood flow without metabolic overload
Food-Based Nutrients for Brain Repair (by food source — not supplement)
These are food sources, not prescriptions. Skip any food you are allergic or sensitive to — multiple sources are listed for each nutrient so alternatives are available.
DHA: wild salmon, sardines, mackerel, pastured egg yolks, fish roe — structural repair of neuronal membranes
Magnesium: pumpkin seeds, dark leafy greens, dark chocolate (unsweetened), legumes, avocado — NMDA receptor blocker; anti-excitotoxic
Choline: egg yolks (pastured), liver, beef — phospholipid synthesis; critical for myelin and membrane repair
Thiamine (B1): pork, liver, sunflower seeds — mitochondrial energy production in neurons
Zinc: oysters, red meat, pumpkin seeds, liver — antioxidant; synaptic signaling repair
Folate (food form): liver, lentils, dark leafy greens, avocado — not folic acid supplement
Stable glucose: protein + fat with every meal, no long gaps — glucose instability is a primary driver of post-concussion cognitive symptoms
CoQ10 from food: beef heart, organ meats — mitochondrial electron transport; neuronal energy production
After Antibiotics — Gut-Brain Rebuild
Raw sauerkraut (refrigerated, not shelf-stable) — 1–2 tablespoons with meals
Kimchi (fermented, not vinegar-based)
Kefir from pasture dairy (if tolerated)
Bone broth from pasture-raised animals
Test — Know Your Baseline
Labs to Request from Your Doctor
RBC Magnesium (not serum)
Full thyroid panel: TSH, free T3, free T4, TPO antibodies
Morning cortisol (8am blood draw) + DHEA-S
Serum iron, ferritin, TIBC, transferrin saturation
Serum copper + ceruloplasmin
Serum zinc
Methylmalonic acid (functional B12 — more sensitive than serum B12)
Homocysteine (elevated = B12/folate/B6 deficiency; neurological risk marker)
Fasting insulin + glucose (HbA1c alone insufficient)
hs-CRP (high-sensitivity C-reactive protein — neuroinflammation marker)
MTHFR gene variants (one-time — determines folic acid safety)
Hormonal panel if symptoms include mood, sleep, or cycle disruption
My change timeline — what I started and when:
Week 1 — I removed:
Week 2 — I added:
What I noticed changing:
Labs I requested (date):
Doctor Visit Prep
Bring this page to your appointment. Fill in before you go. The goal is to use your appointment time efficiently — specific symptoms, specific questions, specific requests.
Appointment Details
Provider name
Appointment date
Appointment type
Current medications + doses
Date of injury
Mechanism of injury
Current Symptoms — Rate Each 0–10 (0 = none, 10 = severe)
Headache
Brain fog / concentration
Light sensitivity
Sound sensitivity
Sleep quality
Mood / irritability / anxiety
Balance / vestibular
Fatigue after activity
Changes since last appointment:
Questions I Want Answered Today
1.
2.
3.
4.
5.
Labs I Am Requesting
Check the ones you want to ask for. Bring this list.
RBC Magnesium
Free T3, Free T4, TSH
Morning cortisol (8am)
DHEA-S
Serum iron, ferritin, TIBC
hs-CRP (neuroinflammation)
Serum copper + ceruloplasmin
Methylmalonic acid (B12)
Homocysteine
Fasting insulin + glucose
MTHFR gene variants
Hormonal panel (if applicable)
Questions my provider has not yet asked — that I want to raise:
Has cervical spine involvement been assessed? My injury involved the neck/upper back.
Has anyone assessed my sleep for airway compromise or sleep-disordered breathing?
My symptoms have a hormonal pattern — can we assess hormone levels in relation to my cycle? (women)
What nutrients do my current medications deplete, and how do we monitor for that over time?
Has the role of dietary excitotoxins in secondary injury been discussed in the context of my recovery?
If I need surgery or dental work, which anesthetics are lower-risk given my TBI history?
My fluoride exposure is significant — has this been considered as a factor in my recovery?
Notes From This Appointment
What my provider said:
Medication or treatment changes:
Labs ordered:
Follow-up date:
What I still need to follow up on:
Daily Symptom Log
Track symptoms, suspected triggers, sleep, and what you changed. Patterns emerge in 2–4 weeks. Bring this to your next appointment.
Rate headache, fog, and mood 1–10 · S = screen-heavy day · E = exercise · M = menstrual · A = antibiotic · Note any food changes or notable exposures
Clean Alternatives
Practical swaps for the highest-burden inputs. Every category here has a documented mechanism for why it matters in TBI recovery.
Water
Natural spring water — findaspring.com
Non-ozonated bottled spring water (Mountain Valley, Tourmaline, Gerolsteiner)
Not RO, not distilled, not tap. Glass or stainless container.
Toothpaste
Tooth powder: baking soda (daily base) + pascalite clay (periodic/acute only — adult use, work with practitioner; contains naturally occurring trace minerals including lead as listed by manufacturer)
Verified clean brands: check EWG Skin Deep, Mamavation, Lead Safe Mama
Avoid: fluoride, NHA (nano-hydroxyapatite), SLS (sodium lauryl sulfate), TiO₂, carrageenan. See: toothpaste.html
Cookware
Cast iron (well-seasoned)
Carbon steel
Stainless steel (18/10)
Glass or enameled cast iron (Le Creuset, Lodge)
Replace nonstick regardless of whether the surface appears damaged — PFAS off-gas at cooking temperatures.
Food Sourcing
Pasture-raised eggs, meat, and dairy — no subtherapeutic antibiotics
Wild-caught fish — not farmed
Organic for EWG Dirty Dozen produce minimum
EWG Dirty Dozen: ewg.org/foodnews
Ingredient Tools
EWG Skin Deep — ewg.org/skindeep — personal care and supplement ingredient database
Mamavation — mamavation.com — PFAS and toxin testing database for food and personal care
Lead Safe Mama — tamararubin.com — heavy metal testing in food, supplements, and household products
Open Food Facts — world.openfoodfacts.org — ingredient scanner; searchable by additive
MSG hidden names list — msg-excitotoxins.html → Hidden Names tab — full three-tier list to bring grocery shopping
Related Pages on This Site
MSG & Excitotoxins — full mechanism on why dietary free glutamate matters specifically for brain injury and TBI recovery
Non-Native EMF — voltage-gated calcium channels, neuroinflammation, and the bedroom environment
Seizures — all brain threshold mechanisms apply; informed consent tab has the full input-by-input breakdown
Dental Care & Neurological Conditions — printable patient information sheet for your dental appointments
Fluoride — enzyme inhibition, mitochondrial disruption, and why fluoride burden matters in recovery
Drug Library — full entry for any medication — includes nutrient depletions, excipients, and recovery considerations
This workbook was prepared using research from theundoctored.com · Allie Johnson, DNM, DIM, PNM · Educational resource — not medical advice.